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This Concept Map, created with IHMC CmapTools, has information related to: Lignocaine, Accelerated repolarisation decreasing the Automaticity and excitability of cells especiallythe Purkinje network, the Kidneys in Urine, Cardiac output and perfusion metabolised by the Liver, Accelerated repolarisation decreasing the Duration of the action potential and effective refractory period, Ventricular fibrillation where Amiodarone can not be used administer 1 - 1.5mg/kg IV slow push Additional dose of 0.5 - 0.75mg/kg may be considered Not to exceed 3.0mg/kg in 24 hrs May be given via ETT, LIGNOCAINE indications Local anaesthetic, Class 1B antiarrhythmic which activates the Sodium channel blockage, 1 - 1.5mg/kg IV slow push Additional dose of 0.5 - 0.75mg/kg may be considered Not to exceed 3.0mg/kg in 24 hrs May be given via ETT acts as a Class 1B antiarrhythmic, Pulseless ventricular tachcardia where Amiodarone can not be used administer 1 - 1.5mg/kg IV slow push Additional dose of 0.5 - 0.75mg/kg may be considered Not to exceed 3.0mg/kg in 24 hrs May be given via ETT, ventricular ectopics which cause VT/VF restoring Normal electrical conduction pathway, Heart rate increasing Cardiac output and perfusion, SA node to pace increasing Cardiac output and perfusion, LIGNOCAINE indications Pulseless ventricular tachcardia where Amiodarone can not be used, the Liver excreted via the Kidneys, Normal electrical conduction pathway encouraging SA node to pace, Sodium channel blockage resulting in Accelerated repolarisation, Normal electrical conduction pathway decreasing Heart rate, LIGNOCAINE indications Ventricular fibrillation where Amiodarone can not be used, Automaticity and excitability of cells especiallythe Purkinje network decreasing ventricular ectopics which cause VT/VF, Duration of the action potential and effective refractory period decreasing ventricular ectopics which cause VT/VF