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This Concept Map, created with IHMC CmapTools, has information related to: GROUP 2 CONCEPT MAP DRAFT (LATEST 9 49AM), Knife-edge Margins PRISTINE GINGIVA, Invasion of the coronal part of the lesion by the Epithelial rete pegs, balance between immune-inflammatory responses in the gingival tissues, chemical mediators such as matrix metalloproteinases, Increased sulcus depth Ingcreased gingival crevicular fluid exudation ESTABLISHED GINGIVITIS, CLINICAL FEATURES seen with Presence of true pocket (ɰmm), Proper Oral Hygiene Practice such as use of mouthwash, Gram Positivee Streptococci and Actinomyces with 15% Gram negative rod species, suclcular area causing possible lacerations, "acquired pellicle" which functions as bacterial attachment, CLINICAL FEATURES include Dark red or magenta in color, MOLECULAR ADSORPTION/ PELLICLE FORMATION composed of salivary glycoproteins, Activation of cytotoxic T-cells produces high levels of GCF exudate, MOLECULAR ADSORPTION/ PELLICLE FORMATION starts automatically 30-60 minutes after toothbrushing, inflammatory infiltrate at the junctional epithelium consists of monocytes/macrophages, CALCULAR DEPOSITS its degree of formation, Early Lesion presents with Invasion of the coronal part of the lesion, Red/Bright Red color Not firm consistency Possible bleeding with gentle probing Bulbous interdental Papilla EARLY GINGIVITIS, experimental state used in animal studies, Mostly plasma cells indicates Chronic inflammation