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This Concept Map, created with IHMC CmapTools, has information related to: GI Week 2 COMBINED - Lipid Digestion, Diagnosis of High Cholesterol has Risk factors, HDL composed of, Lipoproteins derived from VLDLs LDL, cholesterol via LDLR-mediated endocytic uptake uptake by peripheral cells, statins are selective competitive inhibitors for HMG CoA reductase, Lipoproteins generated in liver VLDL, APOB missense mutation results in familial hypercholesterolemia, HMG CoA reductase regulated by cholesterol 27C, major apos Apo B-100 Apo E Apo C-II, squalene 30C cholesterol 27C, HMG CoA reduced by HMG CoA reductase, APOB nonsense mutation results in hypobetalipoproteinemia, function = Transports TAGs, cholesterol, and fat soluble vitamins through the thoracic duct TAGs are hydrolyzed by LPL CM remnants return to liver for degradation, Diffusion then Re-synthesis, Lipids digestion, Lifestyle Control LDL through Diet, Diagnosis of High Cholesterol includes Lipid panel, cholesterol synthesis, ABCA1 Increases HDL, LDL composed of