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This Concept Map, created with IHMC CmapTools, has information related to: GI Week 2 COMBINED (NESTED) - Lipid Digestion, Diagnosis of High Cholesterol has Risk factors, Lipoproteins derived from VLDLs LDL, HDL composed of, Lipoproteins generated in liver VLDL, statins are selective competitive inhibitors for HMG CoA reductase, cholesterol via LDLR-mediated endocytic uptake uptake by peripheral cells, APOB missense mutation results in familial hypercholesterolemia, major apos Apo B-100 Apo E Apo C-II, HMG CoA reductase regulated by cholesterol 27C, HMG CoA reduced by HMG CoA reductase, squalene 30C cholesterol 27C, APOB nonsense mutation results in hypobetalipoproteinemia, function = Transports TAGs, cholesterol, and fat soluble vitamins through the thoracic duct TAGs are hydrolyzed by LPL CM remnants return to liver for degradation, Diffusion then Re-synthesis, Lipids digestion, Lifestyle Control LDL through Diet, cholesterol synthesis, Diagnosis of High Cholesterol includes Lipid panel, LDL composed of, ABCA1 Increases HDL